SOX8
SOX8 (SRY-box 8) هوَ بروتين يُشَفر بواسطة جين SOX8 في الإنسان.[1][2][3]
الوظيفة
![[أيقونة]](https://infokontak.com/ar/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Schepers GE، Bullejos M، Hosking BM، Koopman P (أبريل 2000). "Cloning and characterisation of the Sry-related transcription factor gene Sox8". Nucleic Acids Res. ج. 28 ع. 6: 1473–80. DOI:10.1093/nar/28.6.1473. PMC:111037. PMID:10684944.
- ^ "Entrez Gene: SOX8 SRY (sex determining region Y)-box 8". مؤرشف من الأصل في 2010-12-05.
- ^ Pfeifer D، Poulat F، Holinski-Feder E، Kooy F، Scherer G (أبريل 2000). "The SOX8 gene is located within 700 kb of the tip of chromosome 16p and is deleted in a patient with ATR-16 syndrome". Genomics. ج. 63 ع. 1: 108–16. DOI:10.1006/geno.1999.6060. PMID:10662550.
قراءة متعمقة
- Daniels RJ، Peden JF، Lloyd C، وآخرون (2001). "Sequence, structure and pathology of the fully annotated terminal 2 Mb of the short arm of human chromosome 16". Hum. Mol. Genet. ج. 10 ع. 4: 339–52. DOI:10.1093/hmg/10.4.339. PMID:11157797.
- Cheng YC، Lee CJ، Badge RM، وآخرون (2001). "Sox8 gene expression identifies immature glial cells in developing cerebellum and cerebellar tumours". Brain Res. Mol. Brain Res. ج. 92 ع. 1–2: 193–200. DOI:10.1016/S0169-328X(01)00147-4. PMID:11483257.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Gevaert K، Goethals M، Martens L، وآخرون (2004). "Exploring proteomes and analyzing protein processing by mass spectrometric identification of sorted N-terminal peptides". Nat. Biotechnol. ج. 21 ع. 5: 566–9. DOI:10.1038/nbt810. PMID:12665801.
- Schepers G، Wilson M، Wilhelm D، Koopman P (2003). "SOX8 is expressed during testis differentiation in mice and synergizes with SF1 to activate the Amh promoter in vitro". J. Biol. Chem. ج. 278 ع. 30: 28101–8. DOI:10.1074/jbc.M304067200. PMID:12732652.
{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link) - Ota T، Suzuki Y، Nishikawa T، وآخرون (2004). "Complete sequencing and characterization of 21,243 full-length human cDNAs". Nat. Genet. ج. 36 ع. 1: 40–5. DOI:10.1038/ng1285. PMID:14702039.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The Status, Quality, and Expansion of the NIH Full-Length cDNA Project: The Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Martin J، Han C، Gordon LA، وآخرون (2005). "The sequence and analysis of duplication-rich human chromosome 16". Nature. ج. 432 ع. 7020: 988–94. DOI:10.1038/nature03187. PMID:15616553.
- Wissmüller S، Kosian T، Wolf M، وآخرون (2006). "The high-mobility-group domain of Sox proteins interacts with DNA-binding domains of many transcription factors". Nucleic Acids Res. ج. 34 ع. 6: 1735–44. DOI:10.1093/nar/gkl105. PMC:1421504. PMID:16582099.
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