The predicted log P (XLogP3) of sarpogrelate is 1.2.[12] A 2004 review stated that it was unknown whether sarpogrelate crosses the blood–brain barrier.[13] However, other papers have stated that sarpogrelate minimally crosses into the brain and hence is peripherally selective.[14][15][16] Accordingly, a rat study found that peak sarpogrelate levels were 50-fold lower in the brain and spinal cord than in the circulation.[16][17]
^Pertz H, Elz S (April 1995). "In-vitro pharmacology of sarpogrelate and the enantiomers of its major metabolite: 5-HT2A receptor specificity, stereoselectivity and modulation of ritanserin-induced depression of 5-HT contractions in rat tail artery". The Journal of Pharmacy and Pharmacology. 47 (4): 310–6. doi:10.1111/j.2042-7158.1995.tb05801.x. PMID7791029. S2CID25311518.
^Nishio H, Inoue A, Nakata Y (1996). "Binding affinity of sarpogrelate, a new antiplatelet agent, and its metabolite for serotonin receptor subtypes". Archives Internationales de Pharmacodynamie et de Therapie. 331 (2): 189–202. PMID8937629.
^ abRashid M, Manivet P, Nishio H, Pratuangdejkul J, Rajab M, Ishiguro M, et al. (May 2003). "Identification of the binding sites and selectivity of sarpogrelate, a novel 5-HT2 antagonist, to human 5-HT2A, 5-HT2B and 5-HT2C receptor subtypes by molecular modeling". Life Sci. 73 (2): 193–207. doi:10.1016/s0024-3205(03)00227-3. PMID12738034.
^Pietraszek MH, Takada Y, Taminato A, Yoshimi T, Watanabe I, Takada A (April 1993). "The effect of MCI-9042 on serotonin-induced platelet aggregation in type 2 diabetes mellitus". Thrombosis Research. 70 (2): 131–8. doi:10.1016/0049-3848(93)90154-g. PMID8322284.
^Ogawa S, Takeuchi K, Sugimura K, Sato C, Fukuda M, Lee R, et al. (1999). "The 5-HT2 receptor antagonist sarpogrelate reduces urinary and plasma levels of thromboxane A2 and urinary albumin excretion in non-insulin-dependent diabetes mellitus patients". Clinical and Experimental Pharmacology & Physiology. 26 (5–6): 461–4. doi:10.1111/j.1440-1681.1999.03056.x. PMID10386239. S2CID31041268.{{cite journal}}: CS1 maint: overridden setting (link)
^Rydzewski A, Urano T, Hachiya T, Kaneko H, Baba S, Takada Y, et al. (December 1996). "The effect of a 5HT2 receptor antagonist sarpogrelate (MCI-9042) treatment on platelet function in Buerger's disease". Thrombosis Research. 84 (6): 445–52. doi:10.1016/s0049-3848(96)00212-5. PMID8987165.
^Igarashi M, Okuda T, Oh-i T, Koga M (October 2000). "Changes in plasma serotonin concentration and acceleration plethysmograms in patients with Raynaud's phenomenon after long-term treatment with a 5-HT2 receptor antagonist". The Journal of Dermatology. 27 (10): 643–50. doi:10.1111/j.1346-8138.2000.tb02246.x. PMID11092268. S2CID24884976.
^Kinugawa T, Fujita M, Lee JD, Nakajima H, Hanada H, Miyamoto S (August 2002). "Effectiveness of a novel serotonin blocker, sarpogrelate, for patients with angina pectoris". American Heart Journal. 144 (2): A1–A6. doi:10.1067/mhj.2002.124056. PMID12177659.
^Hayashi T, Sumi D, Matsui-Hirai H, Fukatsu A, Arockia Rani PJ, Kano H, et al. (May 2003). "Sarpogrelate HCl, a selective 5-HT2A antagonist, retards the progression of atherosclerosis through a novel mechanism". Atherosclerosis. 168 (1): 23–31. doi:10.1016/s0021-9150(03)00054-6. PMID12732383.{{cite journal}}: CS1 maint: overridden setting (link)
^Saini HK, Takeda N, Goyal RK, Kumamoto H, Arneja AS, Dhalla NS (2004). "Therapeutic potentials of sarpogrelate in cardiovascular disease". Cardiovasc Drug Rev. 22 (1): 27–54. doi:10.1111/j.1527-3466.2004.tb00130.x. PMID14978517.
^Hashizume H, Kawakami M, Yoshida M, Okada M, Enyo Y, Inomata Y (February 2007). "Sarpogrelate hydrochloride, a 5-HT2A receptor antagonist, attenuates neurogenic pain induced by nucleus pulposus in rats". Spine (Phila Pa 1976). 32 (3): 315–320. doi:10.1097/01.brs.0000253601.35732.c1. PMID17268262.
^Obata H, Saito S, Ishizaki K, Goto F (September 2000). "Antinociception in rat by sarpogrelate, a selective 5-HT(2A) receptor antagonist, is peripheral". Eur J Pharmacol. 404 (1–2): 95–102. doi:10.1016/s0014-2999(00)00522-7. PMID10980267.
^ abNishiyama T (May 2005). "Effects of a 5-HT2A receptor antagonist, sarpogrelate on thermal or inflammatory pain". Eur J Pharmacol. 516 (1): 18–22. doi:10.1016/j.ejphar.2005.04.026. PMID15916757. After oral administration of sarpogrelate to rats, the peak sarpogrelate concentration in the brain and spinal cord was about 2% of the plasma concentration (Komatsu et al., 1991). Thus, sarpogrelate has very low permeability of the blood–brain barrier.