ALYREF
ALYREF (Aly/REF export factor) هوَ بروتين يُشَفر بواسطة جين ALYREF في الإنسان.[1][2][3]
الوظيفة
![[أيقونة]](https://infokontak.com/ar/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Muravenko OV، Gizatullin RZ، Al-Amin AN، Protopopov AI، Kashuba VI، Zelenin AV، Zabarovsky ER (يناير 2001). "Human ALY/BEF gene Map position 17q25.3". Chromosome Res. ج. 8 ع. 6: 562. DOI:10.1023/A:1009236126053. PMID:11032328.
- ^ "Entrez Gene: ALYREF Aly/REF export factor". مؤرشف من الأصل في 2010-03-07.
- ^ "Entrez Gene: THOC4 THO complex 4". مؤرشف من الأصل في 2010-03-07.
قراءة متعمقة
- Bruhn L، Munnerlyn A، Grosschedl R (1997). "ALY, a context-dependent coactivator of LEF-1 and AML-1, is required for TCRalpha enhancer function". Genes Dev. ج. 11 ع. 5: 640–53. DOI:10.1101/gad.11.5.640. PMID:9119228.
- Neubauer G، King A، Rappsilber J، وآخرون (1998). "Mass spectrometry and EST-database searching allows characterization of the multi-protein spliceosome complex". Nat. Genet. ج. 20 ع. 1: 46–50. DOI:10.1038/1700. PMID:9731529.
- Wichmann I، Garcia-Lozano JR، Respaldiza N، وآخرون (1999). "Autoantibodies to transcriptional regulation proteins DEK and ALY in a patient with systemic lupus erythematosus". Hum. Immunol. ج. 60 ع. 1: 57–62. DOI:10.1016/S0198-8859(98)00085-8. PMID:9952027.
- Virbasius CM، Wagner S، Green MR (1999). "A human nuclear-localized chaperone that regulates dimerization, DNA binding, and transcriptional activity of bZIP proteins". Mol. Cell. ج. 4 ع. 2: 219–28. DOI:10.1016/S1097-2765(00)80369-X. PMID:10488337.
- Le Hir H، Izaurralde E، Maquat LE، Moore MJ (2001). "The spliceosome deposits multiple proteins 20-24 nucleotides upstream of mRNA exon-exon junctions". EMBO J. ج. 19 ع. 24: 6860–9. DOI:10.1093/emboj/19.24.6860. PMC:305905. PMID:11118221.
- Kim VN، Kataoka N، Dreyfuss G (2001). "Role of the nonsense-mediated decay factor hUpf3 in the splicing-dependent exon-exon junction complex". Science. ج. 293 ع. 5536: 1832–6. DOI:10.1126/science.1062829. PMID:11546873.
- Luo ML، Zhou Z، Magni K، وآخرون (2001). "Pre-mRNA splicing and mRNA export linked by direct interactions between UAP56 and Aly". Nature. ج. 413 ع. 6856: 644–7. DOI:10.1038/35098106. PMID:11675789.
- Kataoka N، Diem MD، Kim VN، وآخرون (2002). "Magoh, a human homolog of Drosophila mago nashi protein, is a component of the splicing-dependent exon-exon junction complex". EMBO J. ج. 20 ع. 22: 6424–33. DOI:10.1093/emboj/20.22.6424. PMC:125744. PMID:11707413.
- Strässer K، Masuda S، Mason P، وآخرون (2002). "TREX is a conserved complex coupling transcription with messenger RNA export". Nature. ج. 417 ع. 6886: 304–8. DOI:10.1038/nature746. PMID:11979277.
- Jurica MS، Licklider LJ، Gygi SR، وآخرون (2002). "Purification and characterization of native spliceosomes suitable for three-dimensional structural analysis". RNA. ج. 8 ع. 4: 426–39. DOI:10.1017/S1355838202021088. PMC:1370266. PMID:11991638.
- Rappsilber J، Ryder U، Lamond AI، Mann M (2002). "Large-scale proteomic analysis of the human spliceosome". Genome Res. ج. 12 ع. 8: 1231–45. DOI:10.1101/gr.473902. PMC:186633. PMID:12176931.
- Chen IH، Sciabica KS، Sandri-Goldin RM (2002). "ICP27 interacts with the RNA export factor Aly/REF to direct herpes simplex virus type 1 intronless mRNAs to the TAP export pathway". J. Virol. ج. 76 ع. 24: 12877–89. DOI:10.1128/JVI.76.24.12877-12889.2002. PMC:136725. PMID:12438613.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- McCracken S، Longman D، Johnstone IL، وآخرون (2004). "An evolutionarily conserved role for SRm160 in 3'-end processing that functions independently of exon junction complex formation". J. Biol. Chem. ج. 278 ع. 45: 44153–60. DOI:10.1074/jbc.M306856200. PMID:12944400.
{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link) - Pryor A، Tung L، Yang Z، وآخرون (2004). "Growth-regulated expression and G0-specific turnover of the mRNA that encodes URH49, a mammalian DExH/D box protein that is highly related to the mRNA export protein UAP56". Nucleic Acids Res. ج. 32 ع. 6: 1857–65. DOI:10.1093/nar/gkh347. PMC:390356. PMID:15047853.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Ong SE، Mittler G، Mann M (2005). "Identifying and quantifying in vivo methylation sites by heavy methyl SILAC". Nat. Methods. ج. 1 ع. 2: 119–26. DOI:10.1038/nmeth715. PMID:15782174.
- Masuda S، Das R، Cheng H، وآخرون (2005). "Recruitment of the human TREX complex to mRNA during splicing". Genes Dev. ج. 19 ع. 13: 1512–7. DOI:10.1101/gad.1302205. PMC:1172058. PMID:15998806.
- Mertz JA، Kobayashi R، Dudley JP (2007). "ALY is a common coactivator of RUNX1 and c-Myb on the type B leukemogenic virus enhancer". J. Virol. ج. 81 ع. 7: 3503–13. DOI:10.1128/JVI.02253-06. PMC:1866045. PMID:17229714.
- Quaresma AJ، Sievert R، Nickerson JA (2013). "Regulation of mRNA export by the PI3 kinase/AKT signal transduction pathway". Mol. Biol. Cell. ج. 24 ع. 8: 1208–21. DOI:10.1091/mbc.E12-06-0450. PMC:3623641. PMID:23427269.
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