SERPINB7
SERPINB7 (Serpin family B member 7) هوَ بروتين يُشَفر بواسطة جين SERPINB7 في الإنسان.[1]
الوظيفة
![[أيقونة]](https://infokontak.com/ar/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ "Entrez Gene: SERPINB7 serpin peptidase inhibitor, clade B (ovalbumin), member 7". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Tsujimoto M، Tsuruoka N، Ishida N، وآخرون (1997). "Purification, cDNA cloning, and characterization of a new serpin with megakaryocyte maturation activity". J. Biol. Chem. ج. 272 ع. 24: 15373–80. DOI:10.1074/jbc.272.24.15373. PMID:9182567.
{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link) - Miyata T، Nangaku M، Suzuki D، وآخرون (1998). "A mesangium-predominant gene, megsin, is a new serpin upregulated in IgA nephropathy". J. Clin. Invest. ج. 102 ع. 4: 828–36. DOI:10.1172/JCI2450. PMC:508946. PMID:9710452.
- Inagi R، Miyata T، Suzuki D، وآخرون (2001). "Specific tissue distribution of megsin, a novel serpin, in the glomerulus and its up-regulation in IgA nephropathy". Biochem. Biophys. Res. Commun. ج. 286 ع. 5: 1098–106. DOI:10.1006/bbrc.2001.5509. PMID:11527413.
- Inagi R، Miyata T، Imasawa T، وآخرون (2003). "Mesangial cell-predominant gene, megsin". Nephrol. Dial. Transplant. 17 Suppl 9: 32–3. DOI:10.1093/ndt/17.suppl_9.32. PMID:12386281.
- Inagi R، Miyata T، Nangaku M، وآخرون (2003). "Transcriptional regulation of a mesangium-predominant gene, megsin". J. Am. Soc. Nephrol. ج. 13 ع. 11: 2715–22. DOI:10.1097/01.ASN.0000033507.32175.FA. PMID:12397041.
- Strausberg RL، Feingold EA، Grouse LH، وآخرون (2003). "Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences". Proc. Natl. Acad. Sci. U.S.A. ج. 99 ع. 26: 16899–903. DOI:10.1073/pnas.242603899. PMC:139241. PMID:12477932.
- Li YJ، Du Y، Li CX، وآخرون (2004). "Family-based association study showing that immunoglobulin A nephropathy is associated with the polymorphisms 2093C and 2180T in the 3' untranslated region of the Megsin gene". J. Am. Soc. Nephrol. ج. 15 ع. 7: 1739–43. DOI:10.1097/01.ASN.0000130858.00688.46. PMID:15213261.
- Suzuki Y، Yamashita R، Shirota M، وآخرون (2004). "Sequence comparison of human and mouse genes reveals a homologous block structure in the promoter regions". Genome Res. ج. 14 ع. 9: 1711–8. DOI:10.1101/gr.2435604. PMC:515316. PMID:15342556.
- Gerhard DS، Wagner L، Feingold EA، وآخرون (2004). "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)". Genome Res. ج. 14 ع. 10B: 2121–7. DOI:10.1101/gr.2596504. PMC:528928. PMID:15489334.
- Inagi R، Nangaku M، Usuda N، وآخرون (2005). "Novel serpinopathy in rat kidney and pancreas induced by overexpression of megsin". J. Am. Soc. Nephrol. ج. 16 ع. 5: 1339–49. DOI:10.1681/ASN.2004070600. PMID:15788472.
- Xia Y، Li Y، Du Y، وآخرون (2006). "Association of MEGSIN 2093C-2180T haplotype at the 3' untranslated region with disease severity and progression of IgA nephropathy". Nephrol. Dial. Transplant. ج. 21 ع. 6: 1570–4. DOI:10.1093/ndt/gfk096. PMID:16431886.
- Xia YF، Huang S، Li X، وآخرون (2006). "A family-based association study of megsin A23167G polymorphism with susceptibility and progression of IgA nephropathy in a Chinese population". Clin. Nephrol. ج. 65 ع. 3: 153–9. DOI:10.5414/cnp65153. PMID:16550745.
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