AP1M1
AP1M1 (Adaptor related protein complex 1 subunit mu 1) هوَ بروتين يُشَفر بواسطة جين AP1M1 في الإنسان.[1][2]
الوظيفة
![[أيقونة]](https://infokontak.com/ar/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Medigeshi GR، Krikunova M، Radhakrishnan K، Wenzel D، Klingauf J، Schu P (يناير 2008). "AP-1 membrane-cytoplasm recycling regulated by mu1A-adaptin". Traffic. ج. 9 ع. 1: 121–32. DOI:10.1111/j.1600-0854.2007.00672.x. PMID:17988225.
- ^ Peyrard M، Parveneh S، Lagercrantz S، Ekman M، Fransson I، Sahlén S، Dumanski JP (يونيو 1998). "Cloning, expression pattern, and chromosomal assignment to 16q23 of the human gamma-adaptin gene (ADTG)". Genomics. ج. 50 ع. 2: 275–80. DOI:10.1006/geno.1998.5289. PMID:9653655.
قراءة متعمقة
- Geyer M، Fackler OT، Peterlin BM (يوليو 2001). "Structure--function relationships in HIV-1 Nef". EMBO Reports. ج. 2 ع. 7: 580–5. DOI:10.1093/embo-reports/kve141. PMC:1083955. PMID:11463741.
- Bénichou S، Benmerah A (يناير 2003). "[The HIV nef and the Kaposi-sarcoma-associated virus K3/K5 proteins: "parasites"of the endocytosis pathway]". Médecine Sciences. ج. 19 ع. 1: 100–6. DOI:10.1051/medsci/2003191100. PMID:12836198.
- Ohno H، Stewart J، Fournier MC، Bosshart H، Rhee I، Miyatake S، Saito T، Gallusser A، Kirchhausen T، Bonifacino JS (سبتمبر 1995). "Interaction of tyrosine-based sorting signals with clathrin-associated proteins". Science. ج. 269 ع. 5232: 1872–5. DOI:10.1126/science.7569928. PMID:7569928.
- Page LJ، Robinson MS (نوفمبر 1995). "Targeting signals and subunit interactions in coated vesicle adaptor complexes". The Journal of Cell Biology. ج. 131 ع. 3: 619–30. DOI:10.1083/jcb.131.3.619. PMC:2120623. PMID:7593184.
- Seaman MN، Sowerby PJ، Robinson MS (أكتوبر 1996). "Cytosolic and membrane-associated proteins involved in the recruitment of AP-1 adaptors onto the trans-Golgi network". The Journal of Biological Chemistry. ج. 271 ع. 41: 25446–51. DOI:10.1074/jbc.271.41.25446. PMID:8810314.
{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link) - Mangasarian A، Foti M، Aiken C، Chin D، Carpentier JL، Trono D (يناير 1997). "The HIV-1 Nef protein acts as a connector with sorting pathways in the Golgi and at the plasma membrane". Immunity. ج. 6 ع. 1: 67–77. DOI:10.1016/S1074-7613(00)80243-5. PMID:9052838.
- Foti M، Mangasarian A، Piguet V، Lew DP، Krause KH، Trono D، Carpentier JL (أكتوبر 1997). "Nef-mediated clathrin-coated pit formation". The Journal of Cell Biology. ج. 139 ع. 1: 37–47. DOI:10.1083/jcb.139.1.37. PMC:2139808. PMID:9314527.
- Ohno H، Aguilar RC، Fournier MC، Hennecke S، Cosson P، Bonifacino JS (نوفمبر 1997). "Interaction of endocytic signals from the HIV-1 envelope glycoprotein complex with members of the adaptor medium chain family". Virology. ج. 238 ع. 2: 305–15. DOI:10.1006/viro.1997.8839. PMID:9400603.
- Piguet V، Chen YL، Mangasarian A، Foti M، Carpentier JL، Trono D (مايو 1998). "Mechanism of Nef-induced CD4 endocytosis: Nef connects CD4 with the mu chain of adaptor complexes". The EMBO Journal. ج. 17 ع. 9: 2472–81. DOI:10.1093/emboj/17.9.2472. PMC:1170589. PMID:9564030.
- Le Gall S، Erdtmann L، Benichou S، Berlioz-Torrent C، Liu L، Benarous R، Heard JM، Schwartz O (أبريل 1998). "Nef interacts with the mu subunit of clathrin adaptor complexes and reveals a cryptic sorting signal in MHC I molecules". Immunity. ج. 8 ع. 4: 483–95. DOI:10.1016/S1074-7613(00)80553-1. PMID:9586638.
- Craig HM، Pandori MW، Guatelli JC (سبتمبر 1998). "Interaction of HIV-1 Nef with the cellular dileucine-based sorting pathway is required for CD4 down-regulation and optimal viral infectivity". Proceedings of the National Academy of Sciences of the United States of America. ج. 95 ع. 19: 11229–34. DOI:10.1073/pnas.95.19.11229. PMC:21624. PMID:9736718.
- Bresnahan PA، Yonemoto W، Ferrell S، Williams-Herman D، Geleziunas R، Greene WC (نوفمبر 1998). "A dileucine motif in HIV-1 Nef acts as an internalization signal for CD4 downregulation and binds the AP-1 clathrin adaptor". Current Biology. ج. 8 ع. 22: 1235–8. DOI:10.1016/S0960-9822(07)00517-9. PMID:9811606.
- Greenberg M، DeTulleo L، Rapoport I، Skowronski J، Kirchhausen T (نوفمبر 1998). "A dileucine motif in HIV-1 Nef is essential for sorting into clathrin-coated pits and for downregulation of CD4". Current Biology. ج. 8 ع. 22: 1239–42. DOI:10.1016/S0960-9822(07)00518-0. PMID:9811611.
- Owen DJ، Evans PR (نوفمبر 1998). "A structural explanation for the recognition of tyrosine-based endocytotic signals". Science. ج. 282 ع. 5392: 1327–32. DOI:10.1126/science.282.5392.1327. PMID:9812899.
- Berlioz-Torrent C، Shacklett BL، Erdtmann L، Delamarre L، Bouchaert I، Sonigo P، Dokhelar MC، Benarous R (فبراير 1999). "Interactions of the cytoplasmic domains of human and simian retroviral transmembrane proteins with components of the clathrin adaptor complexes modulate intracellular and cell surface expression of envelope glycoproteins". Journal of Virology. ج. 73 ع. 2: 1350–61. PMC:103959. PMID:9882340.
- Fölsch H، Ohno H، Bonifacino JS، Mellman I (أكتوبر 1999). "A novel clathrin adaptor complex mediates basolateral targeting in polarized epithelial cells". Cell. ج. 99 ع. 2: 189–98. DOI:10.1016/S0092-8674(00)81650-5. PMID:10535737.
- Orzech E، Cohen S، Weiss A، Aroeti B (مايو 2000). "Interactions between the exocytic and endocytic pathways in polarized Madin-Darby canine kidney cells". The Journal of Biological Chemistry. ج. 275 ع. 20: 15207–19. DOI:10.1074/jbc.275.20.15207. PMID:10809756.
{{استشهاد بدورية محكمة}}: صيانة الاستشهاد: دوي مجاني غير معلم (link) - Umeda A، Meyerholz A، Ungewickell E (مايو 2000). "Identification of the universal cofactor (auxilin 2) in clathrin coat dissociation". European Journal of Cell Biology. ج. 79 ع. 5: 336–42. DOI:10.1078/S0171-9335(04)70037-0. PMID:10887964.
- Nakagawa T، Setou M، Seog D، Ogasawara K، Dohmae N، Takio K، Hirokawa N (نوفمبر 2000). "A novel motor, KIF13A, transports mannose-6-phosphate receptor to plasma membrane through direct interaction with AP-1 complex". Cell. ج. 103 ع. 4: 569–81. DOI:10.1016/S0092-8674(00)00161-6. PMID:11106728.
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