LYL1
LYL1 (LYL1, basic helix-loop-helix family member) هوَ بروتين يُشَفر بواسطة جين LYL1 في الإنسان.[1][2]
الوظيفة
![[أيقونة]](https://infokontak.com/ar/%D9%85%D9%84%D9%81:Crystal_xedit.svg){kind=small} | هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
الأهمية السريرية
| هذا القسم فارغ أو غير مكتمل. ساهم في توسيعه. (يوليو 2018) |
المراجع
- ^ Mellentin JD، Smith SD، Cleary ML (يوليو 1989). "lyl-1, a novel gene altered by chromosomal translocation in T cell leukemia, codes for a protein with a helix-loop-helix DNA binding motif". Cell. ج. 58 ع. 1: 77–83. DOI:10.1016/0092-8674(89)90404-2. PMID:2752424.
- ^ "Entrez Gene: LYL1 lymphoblastic leukemia derived sequence 1". مؤرشف من الأصل في 2010-12-05.
قراءة متعمقة
- Xia Y، Brown L، Yang CY، Tsan JT، Siciliano MJ، Espinosa R، Le Beau MM، Baer RJ (ديسمبر 1991). "TAL2, a helix-loop-helix gene activated by the (7;9)(q34;q32) translocation in human T-cell leukemia". Proceedings of the National Academy of Sciences of the United States of America. ج. 88 ع. 24: 11416–20. DOI:10.1073/pnas.88.24.11416. PMC:53146. PMID:1763056.
- Kuo SS، Mellentin JD، Copeland NG، Gilbert DJ، Jenkins NA، Cleary ML (يونيو 1991). "Structure, chromosome mapping, and expression of the mouse Lyl-1 gene". Oncogene. ج. 6 ع. 6: 961–8. PMID:2067848.
- Saltman DL، Mellentin JD، Smith SD، Cleary ML (نوفمبر 1990). "Mapping of translocation breakpoints on the short arm of chromosome 19 in acute leukemias by in situ hybridization". Genes, Chromosomes & Cancer. ج. 2 ع. 4: 259–65. DOI:10.1002/gcc.2870020402. PMID:2268576.
- Wadman I، Li J، Bash RO، Forster A، Osada H، Rabbitts TH، Baer R (أكتوبر 1994). "Specific in vivo association between the bHLH and LIM proteins implicated in human T cell leukemia". The EMBO Journal. ج. 13 ع. 20: 4831–9. PMC:395422. PMID:7957052.
- Trask B، Fertitta A، Christensen M، Youngblom J، Bergmann A، Copeland A، de Jong P، Mohrenweiser H، Olsen A، Carrano A (يناير 1993). "Fluorescence in situ hybridization mapping of human chromosome 19: cytogenetic band location of 540 cosmids and 70 genes or DNA markers". Genomics. ج. 15 ع. 1: 133–45. DOI:10.1006/geno.1993.1021. PMID:8432525.
- Miyamoto A، Cui X، Naumovski L، Cleary ML (مايو 1996). "Helix-loop-helix proteins LYL1 and E2a form heterodimeric complexes with distinctive DNA-binding properties in hematolymphoid cells". Molecular and Cellular Biology. ج. 16 ع. 5: 2394–401. DOI:10.1128/mcb.16.5.2394. PMC:231228. PMID:8628307.
- Mahajan MA، Park ST، Sun XH (يونيو 1996). "Association of a novel GTP binding protein, DRG, with TAL oncogenic proteins". Oncogene. ج. 12 ع. 11: 2343–50. PMID:8649774.
- Ferrier R، Nougarede R، Doucet S، Kahn-Perles B، Imbert J، Mathieu-Mahul D (يناير 1999). "Physical interaction of the bHLH LYL1 protein and NF-kappaB1 p105". Oncogene. ج. 18 ع. 4: 995–1005. DOI:10.1038/sj.onc.1202374. PMID:10023675.
- Meng YS، Khoury H، Dick JE، Minden MD (نوفمبر 2005). "Oncogenic potential of the transcription factor LYL1 in acute myeloblastic leukemia". Leukemia. ج. 19 ع. 11: 1941–7. DOI:10.1038/sj.leu.2403836. PMID:16094422.
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